Počet záznamů: 1
Mechanical allodynia and enhanced responses to capsaicin are mediated by PI3K in a paclitaxel model of peripheral neuropathy
SYS 0503873 LBL 01000a^^22220027750^450 005 20240103221918.6 014 $a 85059310372 $2 SCOPUS 014 $a 30471295 $2 PUBMED 014 $a 000457663900017 $2 WOS 017 $a 10.1016/j.neuropharm.2018.11.027 $2 DOI 100 $a 20190411d m y slo 03 ba 101 $a eng 102 $a NL 200 1-
$a Mechanical allodynia and enhanced responses to capsaicin are mediated by PI3K in a paclitaxel model of peripheral neuropathy 215 $a 12 s. 463 -1
$1 001 cav_un_epca*0257317 $1 011 $a 0028-3908 $e 1873-7064 $1 200 1 $a Neuropharmacology $v Roč. 146, Mar 1 (2019), s. 163-174 $1 210 $c Elsevier 610 $a pain 610 $a neuropathy 610 $a chemotherapy 610 $a paclitaxel 610 $a PI3K 700 -1
$3 cav_un_auth*0303144 $a Adámek $b Pavel $p FGU-C $i Funkční morfologie $j Functional Morphology $w Laboratory of Pain Research $T Fyziologický ústav AV ČR, v. v. i. 701 -1
$3 cav_un_auth*0374269 $a Heleš $b Mário $p FGU-C $i Funkční morfologie $j Functional Morphology $w Laboratory of Pain Research $T Fyziologický ústav AV ČR, v. v. i. 701 -1
$3 cav_un_auth*0104306 $a Paleček $b Jiří $p FGU-C $i Funkční morfologie $j Functional Morphology $w Laboratory of Pain Research $z K $T Fyziologický ústav AV ČR, v. v. i. 856 $9 RIV $u https://doi.org/10.1016/j.neuropharm.2018.11.027
Počet záznamů: 1