Nitric oxide signaling pathways

0088973 - FGÚ 2008 RIV SK eng M - Část monografie knihy
Zemančíková, A. - Török, J. - Zicha, Josef - Kuneš, Jaroslav
Inactivation of Gi proteins by pertussis toxin attenuates the adrenergic contractions in isolated arteries from spontaneously hypertensive rats.
[Inaktivace Gi proteinů pertussis toxinem oslabuje adrenergní kontrakce arterií izolovaných ze spontánně hypertenzních potkanů.]
Nitric oxide signaling pathways. Bratislava: Advent-Orion, 2007 - (Pecháňová, O.), s. 83-88. ISBN 978-80-8071-094-1
Grant CEP: GA MŠMT(CZ) 1M0510
Výzkumný záměr: CEZ:AV0Z50110509
Klíčová slova: pertussis toxin * Gi proteins * adrenergic contraction
Kód oboru RIV: FA - Kardiovaskulární nemoci vč. kardiochirurgie

PTX treatment markedly diminishes the effectiveness of adrenergic stimuli in vasculature which could importantly affect the regulation of blood pressure in spontaneously hypertensive rats

PTX treatment markedly diminishes the effectiveness of adrenergic stimuli in vasculature which could importantly affect the regulation of blood pressure in spontaneously hypertensive rats
Trvalý link: http://hdl.handle.net/11104/0150336