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Vasodilator efficiency of endogenous prostanoids, Ca2+-activated K+ channels and nitric oxide in rats with spontaneous, salt-dependent or NO-deficient hypertension

SYSNO ASEP	0364293
Document Type	J - Journal Article
R&D Document Type	Journal Article
Subsidiary J	Článek ve WOS
Title	Vasodilator efficiency of endogenous prostanoids, Ca2+-activated K+ channels and nitric oxide in rats with spontaneous, salt-dependent or NO-deficient hypertension
Author(s)	Behuliak, Michal (FGU-C)_{RID, ORCID} Pintérová, Mária (FGU-C) Kuneš, Jaroslav (FGU-C)_{RID, ORCID} Zicha, Josef (FGU-C)_{RID, ORCID, SAI}
Source Title	Hypertension Research. - : Springer - ISSN 0916-9636 Roč. 34, č. 8 (2011), s. 968-975
Number of pages	8 s.
Language	eng - English
Country	JP - Japan
Keywords	Ca2+-activated K+ channels ; nitric oxide-dependent vasodilatation ; prostacyclin
Subject RIV	ED - Physiology
R&D Projects	GA305/09/0336 GA ČR - Czech Science Foundation (CSF)
	IAA500110902 GA AV ČR - Academy of Sciences of the Czech Republic (AV ČR)
	1M0510 GA MŠMT - Ministry of Education, Youth and Sports (MEYS)
CEZ	AV0Z50110509 - FGU-C (2005-2011)
UT WOS	000293576100014
DOI	10.1038/hr.2011.82
Annotation	Our data suggested that there is a compensatory activation of vasodilator prostanoids and Ca(2+)-activated K(+) channels in rats with experimental hypertension, whereas NO-dependent vasodilatation is not augmented. Thus, the overall activity of vasodilator systems failed to compensate for augmented sympathetic vasoconstriction in hypertensive animals
Workplace	Institute of Physiology
Contact	Lucie Trajhanová, lucie.trajhanova@fgu.cas.cz, Tel.: 241 062 400
Year of Publishing	2012

Number of the records: 1