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TGF-beta regulates Sca-1 expression and plasticity of pre-neoplastic mammary epithelial stem cells

  1. 1.
    SYSNO ASEP0538179
    Document TypeJ - Journal Article
    R&D Document TypeJournal Article
    Subsidiary JČlánek ve WOS
    TitleTGF-beta regulates Sca-1 expression and plasticity of pre-neoplastic mammary epithelial stem cells
    Author(s) Remšík, Jan (BFU-R)
    Pícková, Markéta (BFU-R) ORCID, RID
    Vacek, Ondřej (BFU-R) ORCID
    Fedr, Radek (BFU-R) ORCID
    Binó, Lucia (BFU-R) ORCID
    Hampl, A. (CZ)
    Souček, Karel (BFU-R) RID, ORCID
    Number of authors7
    Source TitleScientific Reports. - : Nature Publishing Group - ISSN 2045-2322
    Roč. 10, č. 1 (2020)
    Number of pages12 s.
    Publication formOnline - E
    Languageeng - English
    CountryGB - United Kingdom
    Keywordsdifferentiation ; identification ; promotes ; state
    Subject RIVBO - Biophysics
    OECD categoryOther biological topics
    R&D ProjectsNV17-28518A GA MZd - Ministry of Health (MZ)
    NV18-08-00245 GA MZd - Ministry of Health (MZ)
    GA20-22984S GA ČR - Czech Science Foundation (CSF)
    Method of publishingOpen access
    Institutional supportBFU-R - RVO:68081707
    UT WOS000548564900110
    EID SCOPUS85087798740
    DOI https://doi.org/10.1038/s41598-020-67827-4
    AnnotationThe epithelial-mesenchymal plasticity, in tight association with stemness, contributes to the mammary gland homeostasis, evolution of early neoplastic lesions and cancer dissemination. Focused on cell surfaceome, we used mouse models of pre-neoplastic mammary epithelial and cancer stem cells to reveal the connection between cell surface markers and distinct cell phenotypes. We mechanistically dissected the TGF-beta family-driven regulation of Sca-1, one of the most commonly used adult stem cell markers. We further provided evidence that TGF-beta disrupts the lineage commitment and promotes the accumulation of tumor-initiating cells in pre-neoplastic cells.
    WorkplaceInstitute of Biophysics
    ContactJana Poláková, polakova@ibp.cz, Tel.: 541 517 244
    Year of Publishing2021
    Electronic addresshttps://doi.org/10.1038/s41598-020-67827-4
Number of the records: 1  

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