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Unique and Common Agonists Act the Insect Juvenile Hormone Rece and the Human AHR

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    SYSNO ASEP0603119
    Document TypeJ - Journal Article
    R&D Document TypeJournal Article
    Subsidiary JČlánek ve WOS
    TitleUnique and Common Agonists Act the Insect Juvenile Hormone Rece and the Human AHR
    Author(s) Sedlák, David (UMG-J) RID
    Tůma, R. (CZ)
    Kolla, Jayaprakash Narayana (UMG-J)
    Mokhamatam, Raveendra Babu (UMG-J)
    Bahrová, Liliia (UMG-J)
    Lisová, Michaela (UMG-J)
    Bittová, Lenka (BC-A) RID, ORCID
    Jindra, Marek (BC-A) RID, ORCID
    Number of authors8
    Article number168883
    Source TitleJournal of Molecular Biology. - : Elsevier - ISSN 0022-2836
    Roč. 437, č. 2 (2025)
    Number of pages19 s.
    Languageeng - English
    CountryNL - Netherlands
    Keywordsaryl-hydrocarbon receptor ; molecular-dynamics ; nuclear receptors ; protein ; modulation ; activation ; disruptors ; tryptophan ; endocrine ; homology
    OECD categoryBiochemistry and molecular biology
    R&D ProjectsGX20-05151X GA ČR - Czech Science Foundation (CSF)
    LM2018130 GA MŠMT - Ministry of Education, Youth and Sports (MEYS)
    Method of publishingOpen access
    Institutional supportUMG-J - RVO:68378050 ; BC-A - RVO:60077344
    UT WOS001375189500001
    EID SCOPUS85211020814
    DOI https://doi.org/10.1016/j.jmb.2024.168883
    AnnotationTranscription factors of the bHLH-PAS family play vital roles in animal development, physiology, and disease. Two members of the family require binding of low-molecular weight ligands for their activity: the vertebrate aryl hydrocarbon receptor (AHR) and the insect juvenile hormone receptor (JHR). In the fly Drosophila melanogaster, the paralogous proteins GCE and MET constitute the ligand-binding component of JHR complexes. Whilst GCE/MET and AHR are phylogenetically heterologous, their mode of action is similar. JHR is targeted by several synthetic agonists that serve as insecticides disrupting the insect endocrine system. AHR is an important regulator of human endocrine homeostasis, and it responds to environmental pollutants and endocrine disruptors. Whether AHR signaling is affected by compounds that can activate JHR has not been reported. To address this question, we screened a chemical library of 50,000 compounds to identify 93 novel JHR agonists in a reporter system based on Drosophila cells. Of these compounds, 26% modulated AHR signaling in an analogous reporter assay in a human cell line, indicating a significant overlap in the agonist repertoires of the two receptors. To explore the structural features of agonist-dependent activation of JHR and AHR, we compared the ligand-binding cavities and their interactions with selective and common ligands of AHR and GCE. Molecular dynamics modeling revealed ligand-specific as well as conserved side chains within the respective cavities. Significance of predicted interactions was supported through site-directed mutagenesis. The results have indicated that (c) 2024 Elsevier Ltd. All rights are reserved, including those for text and data mining, AI training, and similar technologies.
    WorkplaceInstitute of Molecular Genetics
    ContactNikol Škňouřilová, nikol.sknourilova@img.cas.cz, Tel.: 241 063 217
    Year of Publishing2026
    Electronic addresshttps://www.sciencedirect.com/science/article/abs/pii/S0022283624005138?via%3Dihub
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