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Corticosteroids as Selective and Effective Modulators of Glycine Receptors

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    SYSNO ASEP0575132
    Document TypeJ - Journal Article
    R&D Document TypeJournal Article
    Subsidiary JČlánek ve WOS
    TitleCorticosteroids as Selective and Effective Modulators of Glycine Receptors
    Author(s) Solntseva, E. I. (RU)
    Bukanova, J. V. (RU)
    Kondratenko, R. (RU)
    Kudová, Eva (UOCHB-X) RID, ORCID
    Source TitleACS Chemical Neuroscience. - : American Chemical Society - ISSN 1948-7193
    Roč. 14, č. 17 (2023), s. 3132-3142
    Number of pages11 s.
    Languageeng - English
    CountryUS - United States
    KeywordsGABAA receptor ; glycine receptor ; corticosteroids ; structure-activity relationship study
    OECD categoryNeurosciences (including psychophysiology
    R&D ProjectsLX22NPO5104 GA MŠMT - Ministry of Education, Youth and Sports (MEYS)
    Method of publishingOpen access
    Institutional supportUOCHB-X - RVO:61388963
    UT WOS001049417000001
    EID SCOPUS85169054920
    DOI https://doi.org/10.1021/acschemneuro.3c00287
    AnnotationThe mechanism of the negative impact of corticosteroids on the induction and progress of mental illness remains unclear. In this work, we studied the effects of corticosteroids on the activity of neuronal glycine receptors (GlyR) and GABA-A receptors (GABAAR) by measuring the chloride current induced by the application of GABA (2 or 5 μM) to isolated cerebellar Purkinje cells (IGABA) and by the application of glycine (100 μM) to pyramidal neurons of the rat hippocampus (IGly). It was found that corticosterone, 5α-dihydrodeoxycorticosterone, allotetrahydrocorticosterone, cortisol, and 17α,21-dihydroxypregnenolone were able to accelerate the desensitization of the IGly at physiological concentrations (IC50 values varying from 0.39 to 0.72 μM). Next, cortisone, 11-deoxycortisol, 11-deoxycorticosterone, 5β-dihydrodeoxycorticosterone, and tetrahydrocorticosterone accelerated the desensitization of IGly with IC50 values varying from 10.3 to 15.2 μM. Allotetrahydrocorticosterone and tetrahydrocorticosterone potentiated the IGABA albeit with high EC50 values (18–23 μM). The rest of the steroids had no effect on IGABA in the range of concentrations of 1–100 μM. Finally, our study has suggested a structural relationship of the 3β-hydroxyl group/3-oxo group with the selective modulatory activity on GlyRs in contrast to the 3α-hydroxyl group that is pivotal for GABAARs. In summary, our results suggest that increased GlyR desensitization by corticosteroids may contribute to brain dysfunction under chronic stress and identify corticosteroids for further development as selective modulators of GlyRs.
    WorkplaceInstitute of Organic Chemistry and Biochemistry
    Contactasep@uochb.cas.cz ; Kateřina Šperková, Tel.: 232 002 584 ; Jana Procházková, Tel.: 220 183 418
    Year of Publishing2024
    Electronic addresshttps://doi.org/10.1021/acschemneuro.3c00287
Number of the records: 1  

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