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The aryl hydrocarbon receptor: A predominant mediator for the toxicity of emerging dioxin-like compounds

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    SYSNO ASEP0559979
    Document TypeJ - Journal Article
    R&D Document TypeJournal Article
    Subsidiary JČlánek ve WOS
    TitleThe aryl hydrocarbon receptor: A predominant mediator for the toxicity of emerging dioxin-like compounds
    Author(s) Zhang, W. (CN)
    Xie, H. Q. (CN)
    Li, Y. (CN)
    Zhou, Ming-Xi (BC-A) RID, ORCID
    Zhou, Z. (CN)
    Wang, R. (CN)
    Hahn, M. E. (US)
    Zhao, B. (US)
    Number of authors8
    Article number128084
    Source TitleJournal of Hazardous Materials. - : Elsevier - ISSN 0304-3894
    Roč. 426, March 15 (2022)
    Number of pages14 s.
    Languageeng - English
    CountryNL - Netherlands
    KeywordsAryl hydrocarbon receptor ; AHR ligands ; Dioxin-like compounds ; Molecular mechanisms ; Toxicity evaluation
    Subject RIVEF - Botanics
    OECD categoryPlant sciences, botany
    Method of publishingOpen access
    Institutional supportBC-A - RVO:60077344
    UT WOS000752476800005
    EID SCOPUS85121447616
    DOI https://doi.org/10.1016/j.jhazmat.2021.128084
    AnnotationThe aryl hydrocarbon receptor (AHR) is a member of the basic helix-loop-helix/Per-ARNT-Sim (bHLH-PAS) family of transcription factors and has broad biological functions. Early after the identification of the AHR, most studies focused on its roles in regulating the expression of drug-metabolizing enzymes and mediating the toxicity of dioxins and dioxin-like compounds (DLCs). Currently, more diverse functions of AHR have been identified, indicating that AHR is not just a dioxin receptor. Dioxins and DLCs occur ubiquitously and have diverse health/ ecological risks. Additional research is required to identify both shared and compound-specific mechanisms, especially for emerging DLCs such as polyhalogenated carbazoles (PHCZs), polychlorinated diphenyl sulfides (PCDPSs), and others, of which only a few investigations have been performed at present. Many of the toxic effects of emerging DLCs were observed to be predominantly mediated by the AHR because of their structural similarity as dioxins, and the in vitro TCDD-relative potencies of certain emerging DLC congeners are comparable to or even greater than the WHO-TEFs of OctaCDD, OctaCDF, and most coplanar PCBs. Due to the close relationship between AHR biology and environmental science, this review begins by providing novel insights into AHR signaling (canonical and non-canonical), AHR's biochemical properties (AHR structure, AHR-ligand interaction, AHR-DNA binding), and the variations during AHR transactivation. Then, AHR ligand classification and the corresponding mechanisms are discussed, especially the shared and compound-specific, AHR-mediated effects and mechanisms of emerging DLCs. Accordingly, a series of in vivo and in vitro toxicity evaluation methods based on the AHR signaling pathway are reviewed. In light of current advances, future research on traditional and emerging DLCs will enhance our understanding of their mechanisms, toxicity, potency, and ecological impacts.
    WorkplaceBiology Centre (since 2006)
    ContactDana Hypšová, eje@eje.cz, Tel.: 387 775 214
    Year of Publishing2023
    Electronic addresshttps://www.sciencedirect.com/science/article/pii/S0304389421030533?via%3Dihub
Number of the records: 1  

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