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Omega-3 fatty acids promote fatty acid utilization and production of pro-resolving lipid mediators in alternatively activated adipose tissue macrophages

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    SYSNO ASEP0485878
    Document TypeJ - Journal Article
    R&D Document TypeJournal Article
    Subsidiary JČlánek ve WOS
    TitleOmega-3 fatty acids promote fatty acid utilization and production of pro-resolving lipid mediators in alternatively activated adipose tissue macrophages
    Author(s) Rombaldová, Martina (FGU-C) RID, ORCID
    Janovská, Petra (FGU-C) RID, ORCID
    Kopecký, Jan (FGU-C) RID, ORCID
    Kuda, Ondřej (FGU-C) RID, ORCID, SAI
    Source TitleBiochemical and Biophysical Research Communications. - : Elsevier - ISSN 0006-291X
    Roč. 490, č. 3 (2017), s. 1080-1085
    Number of pages6 s.
    Languageeng - English
    CountryUS - United States
    Keywordsadipose tissue ; macrophages ; omega-3 PUFA ; fatty acid re-esterification ; lipolysis ; lipid mediators
    Subject RIVFB - Endocrinology, Diabetology, Metabolism, Nutrition
    OECD categoryEndocrinology and metabolism (including diabetes, hormones)
    R&D ProjectsGA16-05151S GA ČR - Czech Science Foundation (CSF)
    LTAUSA17173 GA MŠMT - Ministry of Education, Youth and Sports (MEYS)
    Institutional supportFGU-C - RVO:67985823
    UT WOS000406820600074
    EID SCOPUS85021702368
    DOI https://doi.org/10.1016/j.bbrc.2017.06.170
    AnnotationIt is becoming increasingly apparent that mutual interactions between adipocytes and immune cells are key to the integrated control of adipose tissue inflammation and lipid metabolism in obesity, but little is known about the non-inflammatory functions of adipose tissue macrophages (ATMs) and how they might be impacted by neighboring adipocytes. In the current study we used metabolipidomic analysis to examine the adaptations to lipid overload of M1 or M2 polarized macrophages co-incubated with adipocytes and explored potential benefits of omega-3 polyunsaturated fatty acids (PUFA). Macrophages adjust their metabolism to process excess lipids and M2 macrophages in turn modulate lipolysis and fatty acids (FA) re-esterification of adipocytes. While M1 macrophages tend to store surplus FA as triacylglycerols and cholesteryl esters in lipid droplets, M2 macrophages channel FA toward re-esterification and beta-oxidation. Dietary omega-3 PUFA enhance beta-oxidation in both M1 and M2. Our data document that ATMs contribute to lipid trafficking in adipose tissue and that omega-3 PUFA could modulate FA metabolism of ATMs.
    WorkplaceInstitute of Physiology
    ContactLucie Trajhanová, lucie.trajhanova@fgu.cas.cz, Tel.: 241 062 400
    Year of Publishing2018
Number of the records: 1  

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