Number of the records: 1  

DNA damage response during mouse oocyte maturation

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    SYSNO ASEP0458917
    Document TypeJ - Journal Article
    R&D Document TypeJournal Article
    Subsidiary JČlánek ve WOS
    TitleDNA damage response during mouse oocyte maturation
    Author(s) Mayer, Alexandra (UZFG-Y) RID
    Baran, Vladimír (UZFG-Y)
    Sakakibara, Y. (JP)
    Brzáková, Adéla (UZFG-Y)
    Ferencová, Ivana (UZFG-Y) ORCID
    Motlík, Jan (UZFG-Y) RID, ORCID
    Kitajima, T. (JP)
    Schultz, R. M. (US)
    Šolc, Petr (UZFG-Y) RID, ORCID
    Source TitleCell Cycle. - : Taylor & Francis - ISSN 1538-4101
    Roč. 15, č. 4 (2016), s. 546-558
    Number of pages13 s.
    Publication formPrint - P
    Languageeng - English
    CountryUS - United States
    Keywordsdouble strand DNA breaks ; DNA damage ; MRE11 ; meiotic maturation ; mouse oocytes
    Subject RIVEB - Genetics ; Molecular Biology
    R&D ProjectsLH12057 GA MŠMT - Ministry of Education, Youth and Sports (MEYS)
    ED2.1.00/03.0124 GA MŠMT - Ministry of Education, Youth and Sports (MEYS)
    Institutional supportUZFG-Y - RVO:67985904
    UT WOS000372110000014
    EID SCOPUS84961285243
    DOI https://doi.org/10.1080/15384101.2015.1128592
    AnnotationBecause low levels of DNA double strand breaks (DSBs) appear not to activate the ATM-mediated prophase I checkpoint in full-grown oocytes, there may exist mechanisms to protect chromosome integrity during meiotic maturation. Using live imaging we demonstrate that low levels of DSBs induced by the radiomimetic drug Neocarzinostatin (NCS) increase the incidence of chromosome fragments and lagging chromosomes but do not lead to APC/C activation and anaphase onset delay. The number of DSBs, represented by gamma H2AX foci, significantly decreases between prophase I and metaphase II in both control and NCS-treated oocytes. Transient treatment with NCS increases >2-fold the number of DSBs in prophase I oocytes, but less than 30% of these oocytes enter anaphase with segregation errors. MRE11, but not ATM, is essential to detect DSBs in prophase I and is involved in H2AX phosphorylation during metaphase I. Inhibiting MRE11 by mirin during meiotic maturation results in anaphase bridges and also increases the number of gamma H2AX foci in metaphase II. Compromised DNA integrity in mirin-treated oocytes indicates a role for MRE11 in chromosome integrity during meiotic maturation.
    WorkplaceInstitute of Animal Physiology and Genetics
    ContactJana Zásmětová, knihovna@iapg.cas.cz, Tel.: 315 639 554
    Year of Publishing2017
Number of the records: 1  

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