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Mechanisms of natural brassinosteroid-induced apoptosis of prostate cancer cells

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    SYSNO ASEP0384624
    Document TypeJ - Journal Article
    R&D Document TypeJournal Article
    Subsidiary JČlánek ve WOS
    TitleMechanisms of natural brassinosteroid-induced apoptosis of prostate cancer cells
    Author(s) Steigerová, J. (CZ)
    Rárová, L. (CZ)
    Oklešťková, Jana (UEB-Q) RID, ORCID, SAI
    Křížová, K. (CZ)
    Levková, M. (CZ)
    Šváchová, M. (CZ)
    Kolář, Z. (CZ)
    Strnad, Miroslav (UEB-Q) RID, ORCID
    Source TitleFood and Chemical Toxicology. - : Elsevier - ISSN 0278-6915
    Roč. 50, č. 11 (2012), s. 4068-4076
    Number of pages9 s.
    Languageeng - English
    CountryGB - United Kingdom
    KeywordsApoptosis ; Brassinosteroids ; Cell cycle
    Subject RIVFD - Oncology ; Hematology
    R&D ProjectsGA301/08/1649 GA ČR - Czech Science Foundation (CSF)
    CEZAV0Z50380511 - UEB-Q (2005-2011)
    UT WOS000310654300027
    DOI https://doi.org/10.1016/j.fct.2012.08.031
    AnnotationBrassinosteroids (BRs) are a group of polyhydroxylated sterol derivatives with important regulatory roles in various plant physiological processes. The aim of this study was to examine the mechanism of the anti-proliferative activity of natural BRs 28-homocastasterone (28-homoCS) and 24-epibrassinolide (24-epiBL) in hormone-sensitive and -insensitive (LNCaP and DU-145, respectively) human prostate cancer cell lines. The effects of BRs on prostate cancer cells were surveyed using flow cytometry, Western blotting, TUNEL, DNA ladder assays and immunofluorescence analyses. The studied BRs inhibited cell growth and induced G(1) blocks in LNCaP cells accompanied by reductions in cyclin D-1, CDK4/6 and pRb expression. Following BR treatment of DU-145 cells, increases in proportions of cells in the G(2)/M phase of cell cycle were observed, accompanied by down-regulation of cyclins A and B-1. Changes in AR localization patterns in LNCaP cells treated with BRs were shown by immunofluorescence analysis. Furthermore, apoptotic detection methods demonstrated induction of apoptosis mediated by BRs in both cell lines, although changes in the expression of apoptosis-related proteins were modulated differently by 28-homoCS and 24-piBL in each cell line. The studied BRs seem to exert potent growth inhibitory and pro-apoptotic effects and could be therefore highly valuable new candidates for prostate anticancer drugs.
    WorkplaceInstitute of Experimental Botany
    ContactDavid Klier, knihovna@ueb.cas.cz, Tel.: 220 390 469
    Year of Publishing2013
Number of the records: 1  

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