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ISL1 is necessary for auditory neuron development and contributes toward tonotopic organization

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    SYSNO ASEP0568863
    Document TypeJ - Journal Article
    R&D Document TypeJournal Article
    Subsidiary JČlánek ve WOS
    TitleISL1 is necessary for auditory neuron development and contributes toward tonotopic organization
    Author(s) Filova, Iva (BTO-N) ORCID
    Pysaněnko, Kateryna (UEM-P) ORCID
    Tavakoli, Mitra (BTO-N)
    Vochyánová, Simona (BTO-N)
    Dvořáková, Martina (BTO-N) ORCID
    Bohuslavová, Romana (BTO-N) RID
    Smolík, Ondřej (BTO-N)
    Fabriciová, Valeria (BTO-N)
    Hrabalová, Petra (BTO-N) RID
    Benešová, Šárka (BTO-N)
    Valihrach, Lukáš (BTO-N) RID, ORCID
    Černý, Jiří (UMG-J)
    Yamoah, E. N. (US)
    Syka, Josef (UEM-P) RID
    Fritzsch, B. (US)
    Pavlínková, Gabriela (BTO-N) RID, ORCID
    Number of authors16
    Article numbere2207433119
    Source TitleProceedings of the National Academy of Sciences of the United States of America. - : National Academy of Sciences - ISSN 0027-8424
    Roč. 119, č. 37 (2022)
    Number of pages12 s.
    Languageeng - English
    CountryUS - United States
    Keywordsspiral ganglion neurons ; auditory nuclei ; inferior colliculus ; auditory maps ; auditory behavior
    Subject RIVFH - Neurology
    OECD categoryNeurosciences (including psychophysiology
    R&D ProjectsGA20-06927S GA ČR - Czech Science Foundation (CSF)
    LM2018129 GA MŠMT - Ministry of Education, Youth and Sports (MEYS)
    EF18_046/0016045 GA MŠMT - Ministry of Education, Youth and Sports (MEYS)
    EF18_046/0015861 GA MŠMT - Ministry of Education, Youth and Sports (MEYS)
    LM2018126 GA MŠMT - Ministry of Education, Youth and Sports (MEYS)
    Method of publishingOpen access
    Institutional supportBTO-N - RVO:86652036 ; UEM-P - RVO:68378041 ; UMG-J - RVO:68378050
    UT WOS000895561900005
    EID SCOPUS85139290572
    DOI10.1073/pnas.2207433119
    AnnotationA cardinal feature of the auditory pathway is frequency selectivity, represented in a tono-topic map from the cochlea to the cortex. The molecular determinants of the auditory frequency map are unknown. Here, we discovered that the transcription factor ISL1 reg-ulates the molecular and cellular features of auditory neurons, including the formation of the spiral ganglion and peripheral and central processes that shape the tonotopic rep-resentation of the auditory map. We selectively knocked out Isl1 in auditory neurons using Neurod1Cre strategies. In the absence of Isl1, spiral ganglion neurons migrate into the central cochlea and beyond, and the cochlear wiring is profoundly reduced and dis-rupted. The central axons of Isl1 mutants lose their topographic projections and segrega-tion at the cochlear nucleus. Transcriptome analysis of spiral ganglion neurons shows that Isl1 regulates neurogenesis, axonogenesis, migration, neurotransmission-related machinery, and synaptic communication patterns. We show that peripheral disorganiza-tion in the cochlea affects the physiological properties of hearing in the midbrain and auditory behavior. Surprisingly, auditory processing features are preserved despite the significant hearing impairment, revealing central auditory pathway resilience and plastic-ity in Isl1 mutant mice. Mutant mice have a reduced acoustic startle reflex, altered prepulse inhibition, and characteristics of compensatory neural hyperactivity centrally. Our findings show that ISL1 is one of the obligatory factors required to sculpt auditory struc-tural and functional tonotopic maps. Still, upon Isl1 deletion, the ensuing central plastic-ity of the auditory pathway does not suffice to overcome developmentally induced peripheral dysfunction of the cochlea.
    WorkplaceInstitute of Biotechnology
    ContactMonika Kopřivová, Monika.Koprivova@ibt.cas.cz, Tel.: 325 873 700
    Year of Publishing2023
    Electronic addresshttps://www.pnas.org/doi/full/10.1073/pnas.2207433119
Number of the records: 1  

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