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Role of oxidative stress in PKC-delta upregulation and cardioprotection induced by chronic intermittent hypoxia

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    0080364 - FGÚ 2007 RIV US eng J - Journal Article
    Kolář, František - Ježková, J. - Balková, P. - Břeh, J. - Neckář, Jan - Novák, F. - Nováková, O. - Tomášová, H. - Srbová, M. - Ošťádal, Bohuslav - Wilhelm, J. - Herget, J.
    Role of oxidative stress in PKC-delta upregulation and cardioprotection induced by chronic intermittent hypoxia.
    [Úloha oxidativního stresu v upregulaci PKC-delta a kardioprotekci vyvolané chronickou intermitentní hypoxií.]
    American Journal of Physiology-Heart and Circulatory Physiology. Roč. 292, č. 1 (2007), H224-H230. ISSN 0363-6135. E-ISSN 1522-1539
    R&D Projects: GA ČR GA305/04/0465
    Grant - others:GA UK(CZ) 153/2005/B-Bio/PrF
    Institutional research plan: CEZ:AV0Z50110509
    Keywords : ischemia-reperfusion * protein kinase C * chronic hypoxia
    Subject RIV: FA - Cardiovascular Diseases incl. Cardiotharic Surgery
    Impact factor: 3.973, year: 2007

    Oxidative stress associated with chronic intermittent hypoxia plays a role in the development of increased cardiac ischemic tolerance. The infarct size-limiting mechanism of chronic intermittent hypoxia involves the PKC-delta-dependent pathway

    Oxidativní stres spojený s adaptací na chronickou intermitentní hypoxii se uplatňuje v indukci zvýšené odolnosti myokardu k ischemicko-reperfuznímu poškození. V mechanizmu omezení rozsahu akutního infarktu myokardu u chronicky hypoxických potkanů hraje úlohu PKC-delta
    Permanent Link: http://hdl.handle.net/11104/0144631

     
     
Number of the records: 1  

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