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Acute effects of ethanol on action potential and intracellular Ca2+ transient in cardiac ventricular cells: a simulation study

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    SYSNO ASEP0453143
    Document TypeJ - Journal Article
    R&D Document TypeJournal Article
    Subsidiary JČlánek ve WOS
    TitleAcute effects of ethanol on action potential and intracellular Ca2+ transient in cardiac ventricular cells: a simulation study
    Author(s) Pásek, Michal (UT-L) RID, ORCID
    Bébarová, M. (CZ)
    Christé, G. (FR)
    Šimurdová, M. (CZ)
    Šimurda, J. (CZ)
    Source TitleMedical & Biological Engineering & Computing. - : Springer - ISSN 0140-0118
    Roč. 54, č. 5 (2016), s. 753-762
    Number of pages10 s.
    Publication formPrint - P
    Languageeng - English
    CountryDE - Germany
    Keywordsethanol ; cardiomyocyte ; action potential ; rat ventricular cell model ; human ventricular cell model
    Subject RIVBO - Biophysics
    Institutional supportUT-L - RVO:61388998
    UT WOS000374470600005
    EID SCOPUS84939446301
    DOI https://doi.org/10.1007/s11517-015-1366-8
    AnnotationAlcohol consumption may result in electrocardiographic changes and arrhythmias, at least partly due to effects of ethanol on cardiac ionic currents. Contractility and intracellular Ca2+ dynamics seem to be altered as well. In this study, we integrated the available (mostly animal) experimental data into previously published models of the rat and human ventricular myocytes to assess the share of ionic current components in ethanol-induced changes in AP configuration and cytosolic Ca2+ transient in ventricular cardiomyocytes. The rat model reproduced well the experimentally observed changes in AP duration (APD) under ethanol (slight prolongation at 0.8 mM and shortening at ≥8 mM). These changes were almost exclusively caused by the ethanol-induced alterations of IK1. The cytosolic Ca2+ transient decreased gradually with the increasing ethanol concentration as a result of the ethanol-induced inhibition of ICa. In the human model, ethanol produced a dose-dependent APD lengthening, dominated by ethanol effect on IKr, the key repolarising current in human ventricles. This effect might contribute to the clinically observed proarrhythmic effects of ethanol in predisposed individuals.
    WorkplaceInstitute of Thermomechanics
    ContactMarie Kajprová, kajprova@it.cas.cz, Tel.: 266 053 154 ; Jana Lahovská, jaja@it.cas.cz, Tel.: 266 053 823
    Year of Publishing2017
Number of the records: 1  

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