Number of the records: 1  

Nitric oxide signaling pathways

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    0088973 - FGÚ 2008 RIV SK eng M - Monography Chapter
    Zemančíková, A. - Török, J. - Zicha, Josef - Kuneš, Jaroslav
    Inactivation of Gi proteins by pertussis toxin attenuates the adrenergic contractions in isolated arteries from spontaneously hypertensive rats.
    [Inaktivace Gi proteinů pertussis toxinem oslabuje adrenergní kontrakce arterií izolovaných ze spontánně hypertenzních potkanů.]
    Nitric oxide signaling pathways. Bratislava: Advent-Orion, 2007 - (Pecháňová, O.), s. 83-88. ISBN 978-80-8071-094-1
    R&D Projects: GA MŠMT(CZ) 1M0510
    Institutional research plan: CEZ:AV0Z50110509
    Keywords : pertussis toxin * Gi proteins * adrenergic contraction
    Subject RIV: FA - Cardiovascular Diseases incl. Cardiotharic Surgery

    PTX treatment markedly diminishes the effectiveness of adrenergic stimuli in vasculature which could importantly affect the regulation of blood pressure in spontaneously hypertensive rats

    PTX treatment markedly diminishes the effectiveness of adrenergic stimuli in vasculature which could importantly affect the regulation of blood pressure in spontaneously hypertensive rats
    Permanent Link: http://hdl.handle.net/11104/0150336

     
     
Number of the records: 1  

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