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EGFRvIII mutations can emerge as late and heterogenous events in glioblastoma development and promote angiogenesis through Src activation
- 1.0467243 - ÚPT 2017 RIV US eng J - Journal Article
Eskilsson, E. - Rosland, G.V. - Talasila, K.M. - Knappskog, S. - Keunen, O. - Sottoriva, A. - Foerster, S. - Solecki, G. - Taxt, T. - Jiřík, Radovan - Fritah, S. - Harter, P.N. - Valk, K. - Al Hossain, J. - Joseph, J.V. - Jahedi, R. - Saed, H.S. - Piccirillo, S.G. - Spiteri, I. - Leiss, L. - Euskirchen, P. - Graziani, G. - Daubon, T. - Lund-Johansen, M. - Enger, P.O. - Winkler, F. - Ritter, C. - Niclou, Simone P. - Watts, C. - Bjerkvig, R. - Miletic, H.
EGFRvIII mutations can emerge as late and heterogenous events in glioblastoma development and promote angiogenesis through Src activation.
Neuro-Oncology. Roč. 18, č. 12 (2016), s. 1644-1655. ISSN 1522-8517. E-ISSN 1523-5866
R&D Projects: GA ČR GAP102/12/2380; GA MŠMT(CZ) LO1212
Institutional support: RVO:68081731
Keywords : angiogenesis * EGFR * EGFRvIII * glioblastoma * invasion
Subject RIV: BH - Optics, Masers, Lasers
Impact factor: 7.786, year: 2016
Background. Amplification of the epidermal growth factor receptor (EGFR) and its mutant EGFRvIII are among the most common genetic alterations in glioblastoma (GBM), the most frequent and most aggressive primary brain tumor.
Permanent Link: http://hdl.handle.net/11104/0265373
Number of the records: 1