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Defective fatty acid uptake in the spontaneously hypertensive rat is a primary determinant of altered glucose metabolism, hyperinsulinemia, and myocardial hypertrophy
- 1.0142161 - FGU-C 20010214 RIV US eng J - Journal Article
Hajri, T. - Ibrahimi, A. - Coburn, C. T. - Knapp jr., F. F. - Kurtz, T. - Pravenec, Michal - Abumrad, N. A.
Defective fatty acid uptake in the spontaneously hypertensive rat is a primary determinant of altered glucose metabolism, hyperinsulinemia, and myocardial hypertrophy.
Journal of Biological Chemistry. Roč. 276, č. 26 (2001), s. 23661-23666. ISSN 0021-9258. E-ISSN 1083-351X
Grant - others:NIH-OER(US) RO1-DK33301; AHA(US) AHA0020639T; AHA(US) AHA0030345T
Institutional research plan: CEZ:AV0Z5011922
Keywords : fatty acid transperter * spontaneously hypertensive rat
Subject RIV: EB - Genetics ; Molecular Biology
Impact factor: 7.258, year: 2001
In this study, we determined that loss of CD36 function in fatty acid transport is a primary determinant in increased glucose uptake by muscle and adipose tissue, hyperinsulinemia, and the heart hypertrophy in the spontaneously hypertensive rat.
Permanent Link: http://hdl.handle.net/11104/0039870
Number of the records: 1